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The O-GlcNAc modification of Gli2 induces its deacetylation and augments transcriptional activity via dissociation from p300, a histone acetyltransferase [60]

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Indeed, upregulated BACH1 levels have been reported in post-mortem PD brains as well as in preclinical disease models and further associated with repression of the expression of NRF2 protective genes (Zhou et al., 2018