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To investigate whether the ROS is generated by the host or the pathogen, we inhibited Bd growth by increasing the temperature beyond the thermal optimum of Bd , and/or supplemented the cells with cysteine (a glutathione precursor) with the hypothesis that if ROS was pathogen generated it should decrease under these conditions

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Interestingly, HGF counteracts the oxidative DNA damage after cerebral ischemia by increasing the level of APE/Ref-1-positive cells in the hippocampal CA1 region (Niimura et al., 2006c)