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Renal biopsy specimens taken from cigarette smokers showed a range of long-term changes with varying degrees of glomerulosclerosis, ischemic glomeruli, interstitial fibrosis and tubular atrophy, and arteriolar hyalinosis [16], This broad spectrum of histologic findings combined with initial renal hyperfiltration suggested that cigarette smoking induced renal damage was primarily the result of alterations of renal hemodynamics and its sequelae on glomerular and tubular function /structure [17,18,19]

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Research indicates that the global emission of microplastics from PCPs can reach up to 12,000 tons annually

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After excluding 971 participants with missing serum cotinine data, 5819 children remained eligible for analysis