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Biomarker studies in patients convalescing from COVID-19 also consistently highlighted the involvement of inflammation and coagulation system dysregulation 32,38

In this regard, it has been proposed that a viral infection in which selfantigens that generate molecular mimicry with myelin proteins cause a loss of tolerance against it, which results in the destruction of myelin mediated by activated T lymphocytes in white matter of the brain and sometimes extending into the gray matter, resulting in defects in the conduction of nerve impulses that leads to symptoms, depending on the affected site of the brain or spinal cord [1] (Figure 1)

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Inflammation and pain: Multiple studies have examined the combinations effects on inflammation and pain

Hossein-nezhad, K