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p53 activation does not noticeably affect GPX4 activity, suggesting that it does not induce ferroptosis via GPX4 ( p53 can be rapidly activated to induce cell cycle arrest when retinal cells are subjected to photodamage, hypoxia, or metabolic stress, enabling DNA repair (83)

Proteins important for vesicular trafficking and vesicle fusion including N-ethylmaleimide (NEM) sensitive factor (NSF) and soluble NSF-attachment proteins (SNAPs) are known to be inactivated by oxidation of key cysteine residues [71,72]

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The pathological process can be defined as the depletion of dopamine-producing neurons located in the substantia nigra pars compacta (SNpc), a region within the midbrain (Dorsey et al., 2018)