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Lastly, by incubating the THP1 cells, treated with the peptides, on a layer of activated endothelial cells (HUVECs activated by LPS), we observed a reduction in cell adhesion, a typical pro-inflammatory mechanism

This has been supported by the presence of autoantibodies (77, 78)

Tarbell's contribution to cardiovascular engineering

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Glutaredoxin GRXS17 associates with the cytosolic iron-sulfur cluster assembly pathway