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Additionally, the formation and deposition of advanced glycation end products (AGEs) are accelerated during DM, which have been shown to promote the generation of ROS to induce mesangial cell apoptosis, thereby playing a crucial role in the pathogenesis of DN [116] (Fig

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It is strongly associated with the development and progression of hepatic steatosis, increased oxidative stress, mitochondrial dysfunction, activation of pro-inflammatory and pro-fibrotic pathways, and systemic metabolic dysregulation