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It has shown potential in: Neutralizing toxic products of lipid peroxidation

4 Treatment DKD is a chronic complication of diabetes, improvements in glycemia control and blood pressure management by blocking the RAAS, currently available therapies could not be able to prevent progression to ESRD completely

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Moreover, NO seems to be the only mediator of cell proliferation arrest through the ERK1/2-p53 signaling pathway ( FIGURE 1 Next to this evidence, in our laboratory it has been demonstrated that GSH depletion modulates the peroxisome proliferator-activated receptor gamma, co-activator 1 alpha (PGC-1) expression and its downstream metabolic pathway in neuronal and skeletal muscle cells ( Figure 1 )