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Since pulmonary T cells are the dominant cell type present in the later phase of acute pulmonary inflammation, the increase in adenosine along with upregulation of A2BAR likely govern the repair process after acute injury ( Prior studies have supported a lung protective role for adenosine signaling through A2BAR

Dr Oelsner said: 'There are anatomic differences in the lung that persist for years after smokers quit and gene activity also remains altered.' The results of the study, which involved academics from a handful of institutions, were published in The Lancet Respiratory Medicine

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