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DSIP is proposed to interact with components of the MAPK cascade via this mechanism providing a potential molecular basis for its glucocorticoid-related biology, anti-proliferative activity, and stress-protective effects

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extrapolation to human physiological outcomes has not been established through controlled human trials

It is at the vanguard of prospective therapeutic treatments for treating metabolic diseases, obesity, and possibly even the cellular mechanisms involved in aging because of its ability to block the enzyme Nicotinamide N-methyltransferase (NNMT)