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The skin-specific mechanisms of GHK-Cu include stimulation of Type I and Type III procollagen synthesis, increased production of dermatan sulfate and other glycosaminoglycans, activation of the enzyme lysyl oxidase (which cross-links newly formed collagen), reduction of matrix metalloproteinase-1 (MMP-1, collagenase) activity, and anti-inflammatory suppression of TNF-alpha-induced IL-6 secretion

When the elimination half-life is minutes, peptide is being cleared roughly as fast as it is absorbed, and the peak plasma concentration achievable is far below what a bolus IV injection produces

Contact our Customer Service Team to receive a form that needs to be completed by your GP to confirm your diagnosis and treatment plan, either by injection or oral tablets

Intravenous N-acetylcystine: the treatment of choice for paracetamol poisoning