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Its rationale lies in modulating immune mechanisms implicated in endometriosis pathophysiology, such as inhibition of TNF- and reduction of inflammatory activation in immune cells

In situ B cell-mediated immune responses and tubulointerstitial inflammation in human lupus nephritis

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Recent research studies indicated that nonphosphorylated CagAs C-terminus has a conserved motif that interacts with the host hepatocyte growth factor receptor to promote inflammation and cellular development by triggering the Akt signalling pathway, which in turn triggers NF-B and -catenin, according to recent studies

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