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For example, in G6PD-deficient individuals, residual hepatic enzyme activity can prove insufficient in stress states where increased G6PD activity is required, as in neonates with comorbid G6PD deficiency and Gilberts syndrome, who are at increased risk for hyperbilirubinemia (96, 97)

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The investigators reported that LR3 showed the lowest IGFBP-binding affinities of the analogs studied while retaining full IGF-1R binding, supporting the molecular model of the bioavailability mechanism