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Nat Rev Immunol 16:581592 Shaulian E, Karin M (2002) AP-1 as a regulator of cell life and death

As discussed above, excessive production of pro-inflammatory mediators and free radicals may cause damage to the nervous tissue, impairing axonal conductivity, causing retrograde neuronal death, and developing the conditions for neuropathic pain [6]

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Poor injection technique can waste peptide, cause unnecessary pain, or result in suboptimal absorption

In ischemic stroke, ferroptosis contributes to neuronal death and brain injury through mechanisms involving iron overload, LOX activation, and system Xc dysfunction