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Description

Both peptides stimulate growth hormone release, but they differ in mechanism, selectivity, and side effect profile

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For energy metabolism, PLB significantly increased ATP levels in KGN cells, suggesting a positive effect on mitochondrial function, which was reversed by YTHDF1 overexpression (Fig

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Chlorogenic acid inhibits the binding of Nrf2 to its repressor protein Keap1 to activate the Nrf2 antioxidant signaling pathway, thereby preventing APAP-induced hepatotoxicity (Wei et al., 2018)

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doi: 10.1038/s41586-021-04221-8 232 RidingRLHarrisJE

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Nonlinearity association between hyperuricemia and all-cause mortality in patients with chronic kidney disease

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