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Although there are recommended therapies for AD, such as acetylcholinesterase inhibitors and the N -methyl-D-aspartate receptor antagonists, they are inefficient and do not prevent disease progression, reflecting our incomplete understanding of AD pathogenesis

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Institutional review boards may require detailed information about research compounds used in approved protocols

Huberman notes that the peptide BPC-157 shows promise in tissue repair, but stresses that more clinical trials are needed to validate its use for longevity