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Moreover, ROS inhibit hepatocyte secretion of VLDL and promote hepatic insulin resistance, inducing liver fat accumulation and necro-inflammation

By converting superoxide anions into hydrogen peroxide, SOD prevents them from reacting with other molecules and causing further damage (97, 98)

The hydroxyl radicals produced via copper-catalyzed Fenton reactions mediate oxidative damage, primarily by oxidizing LDL into oxidized LDL (ox-LDL), which serves as a key driver of atherosclerotic plaque formation [121]

doi: 10.1136/ard-2022-223506 113 PatelS.KhanH.MajumdarA

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